CBD oil or Hemp Oil is an extract derived from cannabis containing a cannabinoid known as “cannabidiol.” More than 80 different cannabinoids have been discovered in cannabis, but CBD is one of the most beneficial.
CBD it is not a typical medical marijuana product. Most medical marijuana products contain high concentrations of another cannabinoid called THC, which stands for “tetrahydrocannabinol.”
THC is the psychoactive substance in cannabis which produces a high. Like all plants, cannabis produces an essential oil. This essential oil can be extracted, allowing for processors to collect concentrated amounts of the herb’s active components.
The cannabis plant is capable of producing over 400 different chemical constituents, cannabis oils are most abundant in a few different types of plant compounds.
These two types are cannabinoids, which are unique to cannabis, and terpenes. Terpenes are the aroma molecules that give cannabis its unique scent. Cannabinoids are chemical compounds found only in cannabis that give the herb its medicinal and wellness benefits.
One of the most abundant cannabinoids is cannabidiol (CBD). CBD is second to it’s more famous sibling, psychoactive tetrahydrocannabinol (THC).
While THC is the compound that causes the classic cannabis “high”, CBD is frequently sought out by those hoping to glean the wellness benefits of cannabis without a mind-altering experience. Indeed, Hemp oil is one of the most potent natural painkillers on the market right now, and can be used to treat numerous different conditions.
Its increasing availability means that it is now easier than ever to find a high-quality CBD oil product and have it delivered right to your door.
You can watch big pharma panic right now as they do everything in their power (lobbyists, disinformation, etc..) to try and prevent the full legalization of cbd. There is a reason for that reaction- they know that CBD can wipe out their profits in the opioid industry.
If you suffer from any of these types of Pain or Disorders, CBD Oil may can help. Anxiety Back Pain Chronic Pain Diabetes Depression Epilepsy Fibromyalgia Insomnia Liver Diseases Migraines Multiple Sclerosis Parkinson’s Disease PTSD Rheumatoid Arthritis
CBD has been used to treat chronic pain symptoms and reduce inflammation. It’s considered to be an alternative to taking addictive opioid prescriptions.
It’s important to note that CBD isn’t yet approved by the Food and Drug Administration (FDA) as a regulated treatment option.
But a 2009 study found that CBD can be used for relieving neuropathic pain. And in two studies, CBD was used to treat inflammatory and neuropathic pain from nerve injury. Researchers found CBD to be an effective alternative treatment because it significantly reduced pain symptoms.
Cannabidiol products are used for the therapeutic benefits without experiencing psychoactive symptoms of medical marijuana. However, it can sometimes be difficult to find a product that only has CBD without THC. In this case, cannabis products that have both CBD and THC can still treat pain effectively.
You can consume CBD in a number of forms including:
- Smoking or vaping. If you want to relieve immediate pain, smoking CBD-rich cannabis is the quickest way to reduce symptoms. Effects can last up to three hours. Smoking or vaping allows you to directly inhale CBD from the cannabis plant, absorbing the chemical into your bloodstream and lungs.
- Edibles. Edibles are foods cooked with the cannabis plant, or cannabis-infused oil or butter. It will take longer to experience symptom relief, but the effects of edibles can last for up to six hours.
- Topicals. CBD cannabis oils can be infused into topical creams and balms to apply directly to the skin. These CBD products can be an effective option for reducing inflammation and helping with external pain.
- Oil extracts. Oils can be applied topically, taken orally, or dissolved under the tongue and absorbed in mouth tissues.
There may be respiratory risks to smoking or vaping marijuana. People with asthma or lung conditions shouldn’t use this method. You should also follow dosage instructions carefully, especially with edibles, to avoid negative side effects of taking too much.
Cannabidiol is thought to be safe with minimal side effects. However, some have noted experiencing the following symptoms after using CBD:
- dry mouth
- slowed digestion
- low blood pressure
Researchers are still testing the effectiveness of CBD to treat chronic pain disorders and further research is needed. There are some success stories with using CBD as a treatment option, but it’s not FDA approved and research has yet to show us the long-term effects of CBD on the body.
Until the FDA approves this treatment, traditional fibromyalgia treatment is recommended.
If you decide to use CBD products for pain management, be sure to consult with a doctor first to help avoid harmful interactions with your current medications and treatments, along with negative side effects.
- Acharya, N., Penukonda, S., Shcheglova, T., Hagymasi, A.T., Basu, S., and Srivastava, P.K. (2017, March 27). Endocannabinoid system acts as a regulator of immune homeostasis in the gut. Proceedings of the National Academy of Sciences of the United States, 114(19), 5005-5010. Retrieved from http://www.pnas.org/content/114/19/5005.full.
- Adhikary, S., Li, H., Heller, J., Skarica, M., Zhang, M., Ganea, D., and Tuma, R.F. (2011). Modulation of Inflammatory Responses by a Cannabinoid-2–Selective Agonist after Spinal Cord Injury. Journal of Neurotrauma, 28(12), 2417–2427. Retrieved from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3235339/.
- Buchweitz, JP., Karmaus, PW., Williams, KJ., Harkema, JR. and Kaminski, NE. (2008, March). Targeted deletion of cannabinoid receptors CB1 and CB2 produced enhanced inflammatory responses to influenza A/PR/8/34 in the absence of presence of Delta9-tetrahydrocannabinol. Journal of Leukocyte Biology, 83(3), 785-96. Retrieved from http://www.jleukbio.org/content/83/3/785.long.
- Clayton, N., Marshall, FH., Bountra, C., and O’Shaughnessy, CT. (2002, April). CB1 and CB2 cannabinoid receptors are implicated in inflammatory pain. Pain, 96(3), 253-60. Retrieved from http://journals.lww.com/pain/pages/articleviewer.aspx?year=2002&issue=04000&article=00005&type=abstract.
- Craft, R.M., Kandasamy, R., and Davis, S.M. (2013, September). Sex differences in anti-allodynic, anti-hyperalgesic and anti-edema effects of Δ9-tetrahydrocannabinol in the rat. Pain, 154(9), 1709-17. Retrieved from http://journals.lww.com/pain/pages/articleviewer.aspx?year=2013&issue=09000&article=00028&type=abstract.
- Croxford, J.L., and Yamamura, T. (2005, September). Cannabinoids and the immune system: potential for the treatment of inflammatory diseases? Journal of Neuroimmunology, 166(1-2), 3-18. Retrieved from http://www.jni-journal.com/article/S0165-5728(05)00160-8/fulltext.
- De Laurentiis, A., Araujo, H.A., and Rettori, V. (2014). Role of the Endocannabinoid System in the Neuroendocrine Responses to Inflammation. Current Pharmaceutical Design, 20(29), 4697-706. Retrieved from http://www.eurekaselect.com/120077/article.
- De Petrocellis, L., Melck, D., Bisogno, T., and Di Marzo, V. (2000, November). Endocannabinoids and fatty acid amides in cancer inflammation and related disorders. Chemistry and Physics of Lipids, 108(1-2), 191-209. Retrieved from http://www.sciencedirect.com/science/article/pii/S0009308400001961.
- Fimiani, C., Liberty, T., Aquirre, AJ., Amin, I., Ali, N. and Stefano, GB. (1999, January). Opiate, cannabinoid, and eicosanoid signaling converges on common intracellular pathways nitric oxide coupling. Prostaglandins & Other Lipid Mediators, 57(1), 23-34. Retrieved from http://www.sciencedirect.com/science/article/pii/S0090698098000689.
- Fukuda, S., Kohsaka, H., Takayasu, A., Yokoyama, W., Miyabe, C., Miyabe, Y., Harigai, M., Miyasaka, N., and Nanki, T. (2014). Cannabinoid receptor 2 as a potential therapeutic target in rheumatoid arthritis. BMC Musculoskeletal Disorders, 15, 275. Retrieved from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4243420/.
- Kinsey, S.G., Mahadevan, A., Zhao, B., Sun, H., Naidu, P.S., Razdan, R.K., Selley, D.E., Imad Damaj, M., and Lichtman, A.H. (2011). The CB2 cannabinoid receptor-selective agonist O-3223 reduces pain and inflammation without apparent cannabinoid behavioral effects. Neuropharmacology, 60(2-3), 244–251. Retrieved from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3021987/.
- Klein, T.W. (2005, May). Cannabinoid-based drugs as anti-inflammatory therapeutics. Nature Reviews Immunology, 5, 400-411. Retrieved from http://www.nature.com/nri/journal/v5/n5/full/nri1602.html.
- Matthews, A.T., and Ross, M.K. (2015). Oxyradical Stress, Endocannabinoids, and Atherosclerosis. Toxics, 3(4), 481–498. Retrieved from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4686160/.
- Michalski, C.W., Maier, M., Erkan, M., Sauliunaite, D., Bergmann, F., Pacher, P., Batkai, S., Giese, N.A., Giese, T., Friess, H., and Kleeff, J. (2008). Cannabinoids Reduce Markers of Inflammation and Fibrosis in Pancreatic Stellate Cells. PLoS ONE, 3(2), e1701. Retrieved from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2253501/.
- Panikashvili, D., Shein, N.A., Mechoulam, R., Trembovler, V, Kohen, R., Alexandrovich, A. and Shohami, E. (2006, May). The endocannabinoid 2-AG protects the blood–brain barrier after closed head injury and inhibits mRNA expression of proinflammatory cytokines. Neurobiology of Disease, 22(2), 257-264. Retrieved from http://www.sciencedirect.com/science/article/pii/S0969996105003074.
- Parker, J., Atez, F., Rossetti, R.G., Skulas, A., Patel, R., and Zurier, R.B. (2008, May). Suppression of human macrophage interleukin-6 by a nonpsychoactive cannabinoid acid. Rheumatology International, 28(7), 631-5. Retrieved from http://link.springer.com/article/10.1007%2Fs00296-007-0489-0.
- Wright, K.L., Duncan, M., and Sharkey, K.A. (2008). Cannabinoid CB2 receptors in the gastrointestinal tract: a regulatory system in states of inflammation. British Journal of Pharmacology, 153(2), 263–270. Retrieved from https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2219529/.